Claim profile
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false
Variants
Sardão, Vilma A.
Sardão, Vilma
Sardão, Vilma
Unidade de I&D
Status
UC Researcher
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3
0
20
0
false
Publications
(All)
Date issued
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Results 1-5 of 5 (Search time: 0.005 seconds).
| Issue Date | Title | Author(s) | Type | Access | |
|---|---|---|---|---|---|
| 1 | 20-Jul-2020 | Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors | Kim, Ha-Neui; Ponte, Filipa; Nookaew, Intawat; Ucer Ozgurel, Serra; Carvalho, Adriana Marques ; Iyer, Srividhya; Warren, Aaron; Aykin-Burns, Nukhet; Krager, Kimberly; Sardão, Vilma A. ; Han, Li; de Cabo, Rafael; Zhao, Haibo; Jilka, Robert L.; Manolagas, Stavros C.; Almeida, Maria | article | openAccess |
| 2 | Dec-2014 | Mitochondrial apoptosis-inducing factor is involved in doxorubicin-induced toxicity on H9c2 cardiomyoblasts | Moreira, Ana C. ; Branco, Ana F. ; Sampaio, Susana F. ; Cunha-Oliveira, Teresa ; Martins, Tatiana R. ; Holy, Jon ; Oliveira, Paulo J. ; Sardão, Vilma A. | article | openAccess |
| 3 | Nov-2013 | Mitochondrial disruption occurs downstream from β-adrenergic overactivation by isoproterenol in differentiated, but not undifferentiated H9c2 cardiomyoblasts: Differential activation of stress and survival pathways | Branco, Ana F. ; Sampaio, Susana F. ; Wieckowski, Mariusz R. ; Sardão, Vilma A. ; Oliveira, Paulo J. | article | openAccess |
| 4 | 2008 | Sanguinarine cytotoxicity on mouse melanoma K1735-M2 cells--Nuclear vs. mitochondrial effects | Serafim, Teresa L. ; Matos, Júlio A. C. ; Sardão, Vilma A. ; Pereira, Gonçalo C. ; Branco, Ana F. ; Pereira, Sandro L. ; Parke, Donna ; Perkins, Edward L. ; Moreno, António J. M. ; Holy, Jon ; Oliveira, Paulo J. | article | openAccess |
| 5 | 2014 | β-adrenergic over-stimulation and cardio-myocyte apoptosis: two receptors, one organelle, two fates? | Branco, Ana F. ; Moreira, Ana C. ; Cunha-Oliveira, Teresa ; Couto, Renata ; Sardão, Vilma A. ; Rizvanov, Albert A. ; Palotas, Andras ; Oliveira, Paulo J. | article | openAccess |