Please use this identifier to cite or link to this item: http://hdl.handle.net/10316/41108
DC FieldValueLanguage
dc.contributor.authorFerreira, André-
dc.contributor.authorSerafim, Teresa L.-
dc.contributor.authorSardão, Vilma A.-
dc.contributor.authorCunha-Oliveira, Teresa-
dc.date.accessioned2017-05-03T20:05:10Z-
dc.date.available2017-05-03T20:05:10Z-
dc.date.issued2015-01-
dc.identifier.urihttp://hdl.handle.net/10316/41108-
dc.description.abstractAbnormal mitochondrial function has long been associated with the development and the progression of cancer. Multiple defects in the mitochondrial genome have been reported for various cancers, however the often disregarded mitochondrial epigenetic landscape provides an additional source of deregulation that may contribute to carcinogenesis.por
dc.language.isoengpor
dc.rightsopenAccesspor
dc.subjectDNA Methylationpor
dc.subjectDNA, Mitochondrialpor
dc.subjectHumanspor
dc.subjectMitochondriapor
dc.subjectNeoplasmspor
dc.subjectEpigenesis, Geneticpor
dc.titleRole of mtDNA-related mitoepigenetic phenomena in cancerpor
dc.typearticle-
degois.publication.firstPage44-9por
degois.publication.lastPage49por
degois.publication.titleEuropean journal of clinical investigationpor
dc.peerreviewedyespor
dc.identifier.doi10.1111/eci.12359por
dc.identifier.doi10.1111/eci.12359-
degois.publication.volume45 Suppl 1por
item.languageiso639-1en-
item.fulltextCom Texto completo-
item.grantfulltextopen-
crisitem.author.deptCNC - Center for Neuroscience and Cell Biology-
crisitem.author.researchunitCNC - Center for Neuroscience and Cell Biology-
crisitem.author.researchunitCNC - Center for Neuroscience and Cell Biology-
crisitem.author.orcid0000-0003-4924-5204-
crisitem.author.orcid0000-0001-7014-4614-
crisitem.author.orcid0000-0002-7382-0339-
Appears in Collections:I&D CNC - Artigos em Revistas Internacionais
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