Please use this identifier to cite or link to this item: https://hdl.handle.net/10316/36952
DC FieldValueLanguage
dc.contributor.authorResende, E. P.-
dc.contributor.authorTodo-Bom, A.-
dc.contributor.authorLoureiro, C.-
dc.contributor.authorMota Pinto, A.-
dc.contributor.authorOliveiros, B.-
dc.contributor.authorMesquita, L.-
dc.contributor.authorSilva, H.C.-
dc.date.accessioned2017-03-06T16:40:10Z-
dc.date.available2017-03-06T16:40:10Z-
dc.date.issued2017-
dc.identifier.issn21735115por
dc.identifier.urihttps://hdl.handle.net/10316/36952-
dc.description.abstractAsthma and rhinitis have a complex etiology, depending on multiple genetic and environmental risk factors. An increasing number of susceptibility genes are currently being identified, but the majority of reported associations have not been consistently replicated across populations of different genetic backgrounds.por
dc.language.isoengpor
dc.rightsopenAccesspor
dc.subjectAsthma; Rhinitis; Allergy; IL4R; IL13; IL17A; GSTP1; Genetic polymorphism; Genetic profilepor
dc.titleAsthma and rhinitis have different genetic profiles for IL13, IL17A and GSTP1 polymorphismspor
dc.typearticle-
degois.publication.firstPage10por
degois.publication.lastPage16por
degois.publication.issue1por
degois.publication.titleRevista Portuguesa de Pneumologia (English Edition)por
dc.peerreviewedyespor
dc.identifier.doi10.1016/j.rppnen.2016.06.009-
dc.identifier.doi10.1016/j.rppnen.2016.06.009por
degois.publication.volume23por
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.openairetypearticle-
item.cerifentitytypePublications-
item.grantfulltextopen-
item.fulltextCom Texto completo-
item.languageiso639-1en-
crisitem.author.researchunitCNC - Center for Neuroscience and Cell Biology-
crisitem.author.orcid0000-0002-1850-6689-
crisitem.author.orcid0000-0002-0820-9568-
crisitem.author.orcid0000-0001-7836-8161-
Appears in Collections:FPCEUC - Artigos em Revistas Nacionais
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